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Descending Modulation and Persistent Pain. Hyperalgesia. Muscle Pain Model, Inflammatory Agents-Induced Secondary hyperalgesia refers to the sensitization that occurs because of changes in spinal cord processing. For example, through a process of central sensitization, the firing of dorsal horn nociceptors can change dramatically in the setting of injury (produced by either tissue or nerve damage).

Secondary hyperalgesia example

AU - Werner, Mads Utke. AU - Mårtensson, Johan. AU - Larsson, Henrik B W. AU - Dahl, Jørgen Berg. PY - 2015. Y1 - 2015 Secondary hyperalgesia occurs in the areas around the injured site because of nociceptor activation in the central nervous system.

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Secondary hyperalgesia occurs when the pain spreads to other areas of the body. Hyperalgesia is defined as "An increased sensitivity to pain, which may be caused by damage to nociceptors or peripheral nerves". It is divided into 2 types, primary and secondary. Primary hyperalgesia - pain and sensitivity in the damaged tissues.

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Hyperalgesia. Muscle Pain Model, Inflammatory Agents-Induced Secondary hyperalgesia refers to the sensitization that occurs because of changes in spinal cord processing. For example, through a process of central sensitization, the firing of dorsal horn nociceptors can change dramatically in the setting of injury (produced by either tissue or nerve damage).

Allyodynia The capsaicin-evoked pain intensity was higher in MCS patients compared with controls (P<0.01, 33 mM). The area of secondary hyperalgesia was larger in both the patient groups compared with controls (P<0.05) at both capsaicin concentrations. Temporal summation was increased in MCS patients compared with controls(P<0.01). Hyperalgesia means increased sensitivity to painful or to normally non-painful stimulation. There are two definitions of “primary” and “secondary” hyperalgesia. (1) According to one definition, a hyperalgesia is primary if it occurs in an inflamed body area or – in mononeuropathies – in the innervation territory of a lesioned nerve.
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Lastly, with a proper schematic diagram, i will try to For example, bad sunburn can cause temporary allodynia, and touching sunburned skin, or running cold or warm water over sunburned skin can be very painful. It is different from hyperalgesia, an exaggerated response from a normally painful stimulus. The term is from Ancient Greek άλλος állos "other" and οδύνη odúnē "pain". The association between areas of secondary hyperalgesia and volumes of the caudate nuclei and other pain relevant brain structures—A 3-tesla MRI study of healthy men. PLoS ONE , 13 (8), [e0201642].

On average, participants reported mild pain ( M = 1.6, SD = 1.1) to mechanical stimulation on normal skin located 10cm from the capsaicin site and these intensity ratings on normal skin were unrelated to life event scores ( p = 0.748). Hyperalgesia is caused either by injury or opioid use.
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AU - Larsson, Henrik B W. AU - Dahl, Jørgen Berg. PY - 2015. Y1 - 2015 Secondary hyperalgesia occurs in the areas around the injured site because of nociceptor activation in the central nervous system. In a new study reported in Pain, scientists from Because secondary hyperalgesia is short-lived, persistent pain after surgery in large part may be primary hyperalgesia, perhaps amplified by central sensitization.

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Madden VJ, Bedwell GJ, Chikezie PC, Rice ASC, Kamerman PR Syst Rev 2019 Aug 19;8(1):208. doi: 10.1186/s13643-019-1120-7. M1 = baseline measurements, including the following: (1) quantification of area of secondary hyperalgesia around heat-capsaicin-stimulated skin on the dominant forearm; (2) brief thermal sensitization on the thigh with 45°C for 3 min followed by quantification of area of secondary hyperalgesia; (3) heat pain detection thresholds (HPDT) in normal skin on the upper arm and in the heat-capsaicin The sample size was based on a power calculation which showed that 25 volunteers were required to achieve 80% power to detect a difference of 20% in area of secondary hyperalgesia, with α=0.05 (two-tailed).